DSpace Repository

Melatonin modulates apoptosis and TRPM2 channels in transfected cells activated by oxidative stress

Show simple item record

dc.creator NAZIROĞLU, Mustafa
dc.creator ÇELİK, Ömer
dc.date 2012-10-09T21:00:00Z
dc.date.accessioned 2020-10-06T10:24:27Z
dc.date.available 2020-10-06T10:24:27Z
dc.identifier 5de3659d-b247-4f3f-b647-8cd38f63fd04
dc.identifier 10.1016/j.physbeh.2012.09.013
dc.identifier https://avesis.sdu.edu.tr/publication/details/5de3659d-b247-4f3f-b647-8cd38f63fd04/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/61287
dc.description Transient receptor potential melastatin-like 2 (TRPM2) is a non-selective Ca2+ permeable cation channel and is known to be activated by H2O2, one of the most important indicators of intracellular oxidative stress. A neurohormone melatonin may have a modulator role on TRPM2 channels activated by oxidative stress because it is a strong antioxidant. In this study we investigated the effects of melatonin on apoptosis, whole cell currents and Ca2+ influx arising from TRPM2 channels activated by H2O2.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Melatonin modulates apoptosis and TRPM2 channels in transfected cells activated by oxidative stress
dc.type info:eu-repo/semantics/article


Files in this item

Files Size Format View

There are no files associated with this item.

This item appears in the following Collection(s)

Show simple item record

Search DSpace


Browse

My Account