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Acquired Bartter-like syndrome association with netilmicin therapy in an extremely low birth weight infant

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dc.creator Ozen, Metehan
dc.creator Akbay, Senay
dc.creator SANDAL, Gonca
dc.date 2014-01-31T22:00:00Z
dc.date.accessioned 2020-10-06T10:15:04Z
dc.date.available 2020-10-06T10:15:04Z
dc.identifier 5b25f4af-90d8-4133-a91e-c9be96f13fed
dc.identifier 10.3109/0886022x.2013.832861
dc.identifier https://avesis.sdu.edu.tr/publication/details/5b25f4af-90d8-4133-a91e-c9be96f13fed/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/61014
dc.description Aminoglycosides are commonly used antibiotics with excellent renal parenchymal penetration. Their clinical effectiveness is counter balanced with the risk of renal toxicity, which develops in a dose-dependent fashion. Aminoglycoside-induced renal tubular dysfunction could result in diffuse damage or manifest as a Fanconi-like syndrome, Bartter-like syndrome (BLS), or distal renal tubular acidosis. 1-4 Although tubulopathy associated with amikacin and gentamicin was reported in adults and rarely children, to the best of our knowledge, netilmicin-associated BLS neither in adults nor in children has been reported in the literature. We here report a 30-week, 770 g male preterm infant who developed BLS just after netilmicin treatment for neonatal sepsis and recovered 6 weeks after the drug cessation.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Acquired Bartter-like syndrome association with netilmicin therapy in an extremely low birth weight infant
dc.type info:eu-repo/semantics/article


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