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Uric Acid is a Useful Tool to Predict Contrast-Induced Nephropathy

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dc.creator MENDİ, Mehmet Ali
dc.creator Afsar, Barış
dc.creator OKSUZ, Fatih
dc.creator TURAK, Osman
dc.creator YAYLA, Cagri
dc.creator Johnson, Richard J.
dc.creator OZCAN, Firat
dc.creator Kanbay, Mehmet
dc.date 2017-07-31T21:00:00Z
dc.date.accessioned 2020-10-06T10:25:50Z
dc.date.available 2020-10-06T10:25:50Z
dc.identifier 68426f56-833f-40d8-bd59-03bdd727049f
dc.identifier 10.1177/0003319716639187
dc.identifier https://avesis.sdu.edu.tr/publication/details/68426f56-833f-40d8-bd59-03bdd727049f/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/62336
dc.description Developing contrast-induced nephropathy (CIN) after primary percutaneous coronary intervention (pPCI) has a negative impact on survival and morbidity. We assessed the predictive value of serum uric acid (SUA) for the development of CIN in patients with ST-segment elevation myocardial infarction (STEMI) who underwent pPCI. Contrast-induced nephropathy was defined an increase of 25% or 0.5 mg/dL in creatinine concentrations within 72 hours after pPCI. Patients were divided into 2 groups according to admission median SUA level. Serum uric acid level was <5.4 mg/dL (group 1; n = 222) and 5.4 mg/dL (group 2; n = 228). Compared to group 1, development of CIN (12% vs 20%, P < .001) was significantly greater in group 2. Using a cut point of >5.45 mg/dL, the SUA level predicted development of CIN with a sensitivity of 70% and specificity of 67%. In multiple logistic regression analysis, SUA level, diabetes mellitus, left ventricular ejection fraction <50%, contrast volume, estimated glomerular filtration rate, and C-reactive protein level emerged as independent predictors of CIN. In conclusion, elevated SUA is an independent risk factor for the development of CIN after pPCI in patients with STEMI.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Uric Acid is a Useful Tool to Predict Contrast-Induced Nephropathy
dc.type info:eu-repo/semantics/article


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