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Fetal pulmonary injury following single high-dose intra-amniotic betamethasone treatment in preterm goat kids

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dc.creator Haligur, Mehmet
dc.creator Antlyali, Meltem
dc.creator SEZİK, Mekin
dc.creator Ozmen, Ozlem
dc.date 2012-11-30T22:00:00Z
dc.date.accessioned 2020-10-06T10:49:07Z
dc.date.available 2020-10-06T10:49:07Z
dc.identifier 9413cc4a-f24f-4c6d-a81e-3ad15e44e9e0
dc.identifier 10.5152/jtgga.2012.38
dc.identifier https://avesis.sdu.edu.tr/publication/details/9413cc4a-f24f-4c6d-a81e-3ad15e44e9e0/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/66684
dc.description Objective: Fetal lung maturation is an extremely important process that is necessary for the survival of the neonates. Conventionally, corticosteroids are administered maternally for inducing fetal lung maturation in preterm fetuses. Alternatively, single-dose intra-amniotic (IA) treatment might be speculated to improve lung maturity. In the goat model, we recently showed that high-dose IA betamethasone (BM) was associated with an increased number of stillbirths and emphysematous changes. The aim of the present study is to expand our previous findings and evaluate the histopathological effects of IA injection of a single high-dose of BM 48 h before induced preterm delivery, using our previously collected specimens.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Fetal pulmonary injury following single high-dose intra-amniotic betamethasone treatment in preterm goat kids
dc.type info:eu-repo/semantics/article


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