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Topiramate modulates hippocampus NMDA receptors via brain Ca2+ homeostasis in pentylentetrazol-induced epilepsy of rats

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dc.creator Vural, Huseyin
dc.creator Yilmaz, Mustafa
dc.creator NAZIROĞLU, Mustafa
dc.creator YÜREKLİ, Vedat Ali
dc.creator Yilmaz, Nigar
dc.creator KUTLUHAN, Süleyman
dc.date 2011-03-31T21:00:00Z
dc.date.accessioned 2020-10-06T11:23:55Z
dc.date.available 2020-10-06T11:23:55Z
dc.identifier cd297576-e8e8-428c-a92d-2e26b971e5b5
dc.identifier 10.3109/10799893.2011.555914
dc.identifier https://avesis.sdu.edu.tr/publication/details/cd297576-e8e8-428c-a92d-2e26b971e5b5/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/72310
dc.description Background: Increase in neuronal Ca2+, activation of hippocampus N-methyl-D-aspartate receptor (NMDAR) and defects in enzymes such as brain cortex microsomal membrane Ca2+-ATPase (MMCA) are thought to play a role in epilepsy. Topiramate (TOP) is a novel drug with broad antiepileptic effect, and its effect on brain cortex MMCA is not known. We investigated effects of TOP on pentylentetrazol (PTZ)-induced MMCA activity and NMDAR subunits in rat brain.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Topiramate modulates hippocampus NMDA receptors via brain Ca2+ homeostasis in pentylentetrazol-induced epilepsy of rats
dc.type info:eu-repo/semantics/article


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