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In vivo evidence suggesting a role for purine-catabolizing enzymes in the pathogenesis of cisplatin-induced nephrotoxicity in rats and effect of erdosteine against this toxicity

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dc.creator Yilmaz, HR
dc.creator Kotuk, M
dc.creator Sogut, S
dc.creator Ozyurt, H
dc.creator Ulu, R
dc.creator Yildirim, Z
dc.date 2004-05-01T00:00:00Z
dc.date.accessioned 2021-12-03T11:46:38Z
dc.date.available 2021-12-03T11:46:38Z
dc.identifier a796720a-1088-4176-a47e-b8f675d843d3
dc.identifier 10.1002/cbf.1069
dc.identifier https://avesis.sdu.edu.tr/publication/details/a796720a-1088-4176-a47e-b8f675d843d3/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/93982
dc.description The aim of this experimental study was to investigate the possible role of adenosine deaminase (AD) and xanthine oxidase (XO) in the pathogenesis of cisplatin-induced nephrotoxicity and the effect of erdosteine in decreasing the toxicity. The intraperitoneal injection of cisplatin (7m kg(-1) body weight) induced a significant increase in plasma creatinine level and blood urea nitrogen (BUN), and plasma and damaged renal tissue activities of AD and XO in rats. Co-treatment with erdosteine (10 mg kg(-1) day(-1)) attenuated the increase in the plasma creatinine and BUN levels, and significantly prevented the increase in tissue and plasma AD and XO activities (P < 0.05). The results of this study revealed that XO and AD may play an important role in the pathogenesis of cisplatin-induced nephrotoxicity. The potent free radical scavenger erdosteine may have protective potential in this process and it will become a promising drug in the prevention of this undesired side-effect of cisplatin. but further studies are needed to illuminate the exact protection mechanism of erdosteine against cisptatin-induced nephrotoxicity. Copyright (C) 2004 John Wiley Sons, Ltd.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title In vivo evidence suggesting a role for purine-catabolizing enzymes in the pathogenesis of cisplatin-induced nephrotoxicity in rats and effect of erdosteine against this toxicity
dc.type info:eu-repo/semantics/article


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