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CDK4/6 inhibitors: mechanisms of resistance and potential biomarkers of responsiveness in breast cancer

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dc.creator ÇETİN, Bülent
dc.creator Wabl, Chiara A.
dc.creator Gumusay, Ozge
dc.date 2022-02-01T00:00:00Z
dc.date.accessioned 2022-05-10T11:18:38Z
dc.date.available 2022-05-10T11:18:38Z
dc.identifier 63e3dab0-05d4-464a-87a0-c09c9e4774c1
dc.identifier 10.2217/fon-2021-0842
dc.identifier https://avesis.sdu.edu.tr/publication/details/63e3dab0-05d4-464a-87a0-c09c9e4774c1/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/96671
dc.description Hormone receptor (HR)-positive, HER2-negative tumors represent the most common form of metastatic breast cancer (MBC), and endocrine therapy has been the mainstay treatment for several decades. Recently, a novel drug class called CDK4/6 inhibitors in combination with endocrine therapy have remarkably improved the outcome of patients with HR-positive, HER2-negative MBC by targeting the cell cycle machinery and overcoming aspects of endocrine resistance. Several potential cell-cycle-specific and nonspecific mechanisms of resistance to CDK4/6 inhibitors have been reported in recent studies. This review discusses potential resistance mechanisms to CDK4/6 inhibitors, the use of biomarkers to guide treatment for HR-positive, HER2-negative MBC and possible approaches to overcome resistance to CDK4/6 inhibitors.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title CDK4/6 inhibitors: mechanisms of resistance and potential biomarkers of responsiveness in breast cancer
dc.type info:eu-repo/semantics/article


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