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Serum Zonulin, Claudin-5, junction Adhesion Molecule-A, Occludin and Tricellulin levels in children with autism spectrum disorder

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dc.creator Arıcan, Hülya Mete
dc.creator IŞIK, Ümit
dc.creator Eroğlu, Havvanur
dc.creator BÜYÜKBAYRAM, Halil İbrahim
dc.date 2023-01-01T00:00:00Z
dc.date.accessioned 2025-02-25T10:19:31Z
dc.date.available 2025-02-25T10:19:31Z
dc.identifier 3178594c-28d9-4c75-97de-6841ba5d2440
dc.identifier 10.1080/20473869.2023.2298563
dc.identifier https://avesis.sdu.edu.tr/publication/details/3178594c-28d9-4c75-97de-6841ba5d2440/oai
dc.identifier.uri http://acikerisim.sdu.edu.tr/xmlui/handle/123456789/99258
dc.description Introduction: The aim of this study is to determine whether there is a difference in serum Zonulin, Occludin, Claudin-5, Junctional Adhesion Molecule-A (JAM-A), Tricellulin levels between individuals diagnosed with Autism Spectrum Disorder (ASD) and healthy controls. Method: A total of 40 children with ASD and 40 controls aged 2-12 years were enrolled in this study. Serum levels of biochemical variables were determined using commercial enzyme-linked immunosorbent assay kits. Childhood Autism Rating Scale, Autism Behavior Checklist and Aberrant Behavior Checklist were applied to determine the severity of autistic symptoms. Results: Serum Zonulin, Occludin, Claudin-5, Tricellulin and JAM-A levels did not differ between the study and control groups. There was no relationship between the severity of ASD symptoms and biochemical parameters. Discussion: Although the findings of the study suggest that the permeability of the Blood Brain Barrier (BBB) is not effective in the etiology of ASD, there is a need for new studies addressing the limitations of our study in a larger and heterogeneous sample in order to generalize the findings.
dc.language eng
dc.rights info:eu-repo/semantics/closedAccess
dc.title Serum Zonulin, Claudin-5, junction Adhesion Molecule-A, Occludin and Tricellulin levels in children with autism spectrum disorder
dc.type info:eu-repo/semantics/article


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